Alright, so the dystrophin gene is a huge gene on the X-chromosome, that has 79 exons and is over 2 million base pairs in length.

抗肌萎缩蛋白基因是X染色体上一个庞大的基因，含有79个外显子，全长超过2000kbp（书上通常缩写为bp，碱基对）。

Now, the fact that both Duchenne and Becker muscular dystrophy result from mutations in the same dystrophin gene means that they are " allelic disorders, "

DMD和BMD都是由于同一抗肌萎缩蛋白基因突变引起的，意味着二者是“等位基因疾病”。

Within that group, dystrophinopathies are the most common, which includes Duchenne muscular dystrophy, or DMD, and Becker muscular dystrophy, both of which result from mutations in the dystrophin gene.

这些功能紊乱中，营养不良疾病是最常见的，包括Duchenne型肌营养不良（DMD）和Becker型肌营养不良（BMD）；这二者都是由抗肌萎缩蛋白基因突变导致的。

Now if this inactivation's random, you'd expect about half of the female's cells to have a functional dystrophin gene and the other half to have a defective dystrophin gene, and these people are typically asymptomatic.

如果失活是随机的，可以预料到女性一半的细胞中抗肌萎缩蛋白基因是正常的，另一半是有缺陷的，通常这些人无临床症状。

Having said that, if more cells end up with the defective dystrophin gene, and less with the functional one, they can end up being " manifesting carriers, " meaning that they manifest or show some symptoms.

话虽如此，如果机体中有该基因缺陷的细胞更多，而正常基因更少，这一类人群就是“显性携带者”，意味着他们也会有部分症状表现。