In primary hyperaldosteronism, the main problem is that zona glomerulosa cells secrete high levels of aldosterone, and that aldosterone has a negative feedback effect on renin, and so it actually inhibits renin production.

在原发性醛固酮增多症中，主要问题是球状带细胞分泌高水平的醛固酮；醛固酮对肾素有负反馈作用，因而实际上抑制肾素生成。

All that being said, in situations where somebody produces too much aldosterone, like primary hyperaldosteronism, then there's more potassium secretion by the principal cells and excreted, and therefore less potassium is retained, leading to hypokalemia.

说了这么多，在某人产生了太多醛固酮的情况下，比如原发性醛固酮增多症，更多的钾被主细胞分泌并排泄，这意味着更少的钾被保留，导致了低钾血症。