Professor Se-Jin Lee began modifying the myostatin in mice in the 1990s.

李世进教授在二十世纪九十年代开始对小鼠的肌肉生长抑制素进行研究调节。

They said they lack just a single gene and that's the gene that encodes myostatin.

他们说这些老鼠只缺少一个基因，即编码肌肉生长抑素的基因。

The protein follistatin, has an antagonist effect to myostatin, diminishing myostatin's muscle limiting effect.

卵泡抑素蛋白与肌抑素存在拮抗作用，它能降低肌抑素对肌肉的限制。

The more myostatin present, the lower the amount of muscle you can build.

肌抑素越多，肌肉量越多。

As seen in other animals, the lack of myostatin can invoke unnatural amounts of muscle.

此亦见于其他动物中，肌抑素减少会导致肌肉量的不正常增长。

The function of the myostatin gene was discovered by researchers in 1997.

研究人员于 1997 年发现了肌肉生长抑制素基因的功能。

The gene that held the mutation is associated with the production of myostatin.

发生突变的基因与肌肉生长抑制素的产生有关。

Clearly, there is more to consider than simply stopping myostatin.

显然，除了简单地停止肌肉生长抑制素之外， 还有更多需要考虑的因素。

Fittingly, if your goal is to build muscle, you want myostatin reduced, but not to the point of completely removing it.

若你的目标是健身增肌，你会希望减低肌抑素但又不想完全消除它。

With roughly 150 milligrams of epicatechin supplementation per day, the 6 middle-aged subjects saw a 16.6% myostatin decrease and a 49.2% follistatin increase.

每日 150 毫克的表儿茶素补剂，使六位中年实验者的肌抑素降低 16.6%，卵泡抑素则增长了 49.2%。

Regeneron, an American pharma company, is testing drugs that block myostatin and activin, proteins that inhibit muscle growth in the body.

美国制药公司 Regeneron 正在测试阻断肌生长抑制素和激活素（两种抑制人体肌肉生长的蛋白质）的药物。

Rather than beefing up an athlete's muscles, another new class of drugs, myostatin inhibitors, slow the routine breakdown of muscle tissue, offering an alternative way of increasing strength.

另一类新药，“肌抑素抑制剂”，它无法增强运动员的肌肉，而是通过减缓肌肉组织的常规分解来增强力量。

And a 2007 study on mice found that inhibiting myostatin increased muscle mass, as we’d expect, but didn’t make those mice any stronger than the others.

2007 年对小鼠的一项研究发现， 抑制肌肉生长抑制素会增加肌肉质量， 正如我们预期的那样，但并没有使这些小鼠比其他小鼠更强壮。