Abeta tends to misfold and become sticky, eventually clumping together to form soluble oligomers.

Abeta 倾向于错误折叠并变得粘稠，最终聚集在一起形成可溶性低聚物。

There they make healthy Tau proteins start to misfold as well, spreading pathology across the brain.

在那里， 它们使健康的 Tau 蛋白也开始错误折叠，从而在整个大脑中传播病理学。